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A recent medical study reveals a molecular link connecting a common type of human papillomavirus, from the (beta) family, to the development of squamous cell carcinoma in populations most susceptible to immune deficiency. The genetic analysis shows that the virus is capable of integrating into the cell's genome, disrupting DNA repair mechanisms and enhancing regulated division pathways, indicating a potential causal role that goes beyond the previous belief that it is merely an auxiliary factor to ultraviolet damage. The results demonstrate that detecting viral integration signatures in tumor samples may guide doctors towards more accurate diagnoses and open the door to therapeutic interventions that directly target the viral component in highly susceptible patients. Researchers also call for broader studies to determine the prevalence of this pattern in skin cancers and to assess the feasibility of including it in early detection screenings and post-treatment monitoring. The study emphasizes that prevention remains centered around reducing exposure to ultraviolet radiation; however, understanding the molecular structure of tumors in some patients adds a new layer to selective prevention and enhances the consideration of vaccines and targeted strategies against beta patterns in the most vulnerable groups. The findings conclude that combining traditional prevention with genetic testing may increase survival chances and reduce the long-term burden of skin cancer.